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Mechanisms linking environmental agents to the development of Autoimmune Diseases Oxidative stress-mediated disturbance of Nrf2, induction of NF-kB, PARP1, and NLRP3 can cause activation of both innate and adaptive immune systems, resulting in pro-inflammatory cytokines and production of autoantibodies, leading to tissue damage in Autoimmune disease

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The overall reaction for the formation of • O 2 − from NADPH is as follows (Figure 15): If NOX expression is not properly regulated, NOX-associated ROS can promote OS, aberrant signaling, and genomic instability [229]